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[General] Test ABMM Sample Questions - New Exam ABMM Braindumps

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【General】 Test ABMM Sample Questions - New Exam ABMM Braindumps

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American Society of Microbiology ABMM Sample Questions (Q86-Q91):NEW QUESTION # 86
Acyclovir is a commonly used antiviral agent effective against Herpes Simplex Virus (HSV) and Varicella- Zoster Virus (VZV). Its selective toxicity relies on which initial step for activation within infected cells?
  • A. Blocking viral entry through competitive inhibition of cellular receptors
  • B. Direct inhibition of viral RNA polymerase activity
  • C. Phosphorylation by host cell mitochondrial kinases
  • D. Monophosphorylation primarily by viral thymidine kinase
Answer: D

NEW QUESTION # 87
A patient with HIV infection and a very low CD4+ T-cell count develops a disseminated infection with a fungus that is commonly found in pigeon droppings. The MOST likely causative agent is:
  • A. Candida glabrata
  • B. Saccharomyces cerevisiae
  • C. Cryptococcus gattii
  • D. Cryptococcus neoformans
Answer: D

NEW QUESTION # 88
A sputum sample from a patient with suspected pulmonary tuberculosis is submitted for AFB smear and culture. The smear is reported as positive for acid-fast bacilli (4+). Which method is considered the gold standard for confirming the identification of Mycobacterium tuberculosis complex from culture?
  • A. HPLC analysis of mycolic acids
  • B. Nucleic acid amplification test (NAAT) targeting specific MTC sequences
  • C. Growth rate and colony morphology on Löwenstein-Jensen media
  • D. Niacin accumulation test
Answer: B

NEW QUESTION # 89
A clinical isolate of Klebsiella pneumoniae from a patient with a refractory urinary tract infection exhibits resistance to carbapenems (imipenem, meropenem, ertapenem). PCR analysis reveals the presence of the blaKPC gene. Which of the following enzymatic mechanisms is responsible for this resistance?
  • A. Trapping of the carbapenem molecule by a decoy protein.
  • B. Modification of the ribosomal target site, preventing antibiotic binding.
  • C. Efflux of the carbapenem antibiotic out of the bacterial cell.
  • D. Hydrolysis of the beta-lactam ring, leading to an inactive antibiotic derivative.
Answer: D

NEW QUESTION # 90
A clinical microbiology laboratory is monitoring the emergence of resistance to colistin in Gram-negative bacteria. Resistance to colistin is often mediated by modifications of the bacterial lipopolysaccharide (LPS).
The genetic basis for acquired colistin resistance most commonly involves:
  • A. Mutations in genes encoding enzymes that modify the lipid A component of LPS.
  • B. Acquisition of genes encoding efflux pumps that specifically target colistin.
  • C. Alterations in the outer membrane porin proteins, reducing colistin uptake.
  • D. Production of enzymes that directly inactivate the colistin molecule.
Answer: A

NEW QUESTION # 91
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