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[General] Latest EFM Test Testking | High Pass-Rate EFM: Certified - Electronic Fetal Moni

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【General】 Latest EFM Test Testking | High Pass-Rate EFM: Certified - Electronic Fetal Moni

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NCC Certified - Electronic Fetal Monitoring Sample Questions (Q101-Q106):NEW QUESTION # 101
The most highly oxygenated blood in the fetal circulation is found in the
  • A. descending aorta
  • B. pulmonary arteries
  • C. ductus venosus
Answer: C
Explanation:
Comprehensive and Detailed Explanation From Exact Extract Sources:
In fetal physiology, the highest oxygen saturation exists in the umbilical vein, which then flows through the ductus venosus before entering the right atrium.
According to Creasy & Resnik Maternal-Fetal Medicine, and AWHONN physiologic foundations:
* The umbilical vein carries oxygen-rich blood from the placenta (approx. 80% saturation).
* Most of this blood bypasses the liver via the ductus venosus, which therefore contains the most highly oxygenated blood within the fetal circulatory system.
By contrast:
* The descending aorta contains mixed blood with significantly lower oxygen content due to mixing after passage through the ductus arteriosus.
* The pulmonary arteries in the fetus carry predominantly deoxygenated blood, since fetal lungs are fluid-filled and have high pulmonary vascular resistance.
Thus, the structure containing the highest fetal oxygen concentration is the ductus venosus.
References:Creasy & Resnik - Maternal Fetal Medicine;AWHONN Fetal Monitoring;Simpson & Miller - Fetal Monitoring Physiology;NCC C-EFM Content Outline - Physiology Domain.

NEW QUESTION # 102
The most probable underlying fetal physiologic cause for this tracing would be:

  • A. Vagal nerve stimulation in response to hypoxemia
  • B. Myocardial hypoxic depression
  • C. Release of catecholamines
Answer: C
Explanation:
Comprehensive and Detailed Explanation From NCC-Aligned Sources:
This tracing shows:
* Baseline ~145 bpm
* Minimal variability
* No accelerations or decelerations
* Very little fluctuation # resembles a flat/minimal variability Category II tracing The key physiologic mechanism behind minimal variability in the presence of a normal baseline and normal contraction pattern is most often:
Increased fetal sympathetic tone, driven by catecholamine release (epinephrine and norepinephrine).
NCC and AWHONN explain:
* Catecholamine release (due to fetal stress, early hypoxemia, or maternal stress) results in:
* Reduced beat-to-beat fluctuation
* Minimal baseline variability
* This is considered an early compensatory mechanism, not yet a decompensated hypoxic state.
Why the other answers are incorrect:
* A. Myocardial hypoxic depression
* Causes absent variability, NOT minimal variability.
* Represents advanced or severe hypoxia. The FHR here is not absent variability.
* C. Vagal stimulation in response to hypoxemia
* Produces decelerations, especially late or prolonged.
* This strip shows no decelerations, ruling this out.
Therefore the most accurate physiologic explanation is B. Release of catecholamines.
References:NCC C-EFM Candidate Guide; AWHONN FHMPP; NICHD Baseline Variability Definitions; Menihan EFM; Simpson & Creehan; Creasy & Resnik.

NEW QUESTION # 103
(Full question)
Spontaneous fetal heart rate accelerations indicate
  • A. integrated response of the fetal central nervous system
  • B. immaturity of the fetal parasympathetic nervous system
  • C. dominance of the fetal sympathetic nervous system
Answer: A
Explanation:
Comprehensive and Detailed Explanation From Exact Extract (No URLs):
NCC references (AWHONN, Menihan, Simpson, Creasy & Resnik) consistently state that fetal accelerations are a reassuring sign of intact neurologic function. Accelerations represent the interaction of both the sympathetic and parasympathetic branches moderated through the central nervous system, reflecting effective autonomic regulation.
AWHONN specifically describes fetal accelerations as:
* A maturity marker of CNS function,
* Reflecting vigorous fetal movement,
* Demonstrating adequate oxygenation,
* Indicating a well-oxygenated brainstem and cortex.
Simpson & Miller emphasize that accelerations require both systems to be functioning and respond appropriately, which confirms CNS integration, not sympathetic or parasympathetic dominance alone.
Therefore, spontaneous accelerations indicate an integrated CNS response, making Option C the correct NCC-aligned answer.

NEW QUESTION # 104
A woman is being induced with oxytocin. The tracing shown is representative of 20 minutes. Based on this tracing, the next step would be to:

  • A. Discontinue oxytocin
  • B. Place a spiral electrode
  • C. Proceed to operative birth
Answer: A
Explanation:
Comprehensive and Detailed Explanation From Exact Extract-Based NCC C-EFM References:
Evaluation of a tracing during oxytocin induction requires analysis of fetal status (baseline, variability, accelerations, decelerations) and uterine activity, with attention to tachysystole and fetal intolerance. NCC, AWHONN, Miller, Menihan, Simpson, and the NICHD guidelines all emphasize that oxytocin must be adjusted based on fetal response and contraction frequency.
Baseline:
The fetal heart rate baseline is approximately 150 bpm, which is within the normal range of 110-160 bpm.
Variability:
The tracing shows minimal variability (approximately 1-4 bpm amplitude). Minimal variability for a sustained period is categorized as a Category II pattern under NCC/NICHD classification.
Accelerations:
No accelerations are present during the 20-minute representative segment.
Decelerations:
There are no recurrent variable, no recurrent late, and no prolonged decelerations.
Uterine Activity:
The tracing shows very frequent contractions-approximately every 1½ to 2 minutes, which meets the NCC definition of tachysystole when averaged over 10 minutes (more than 5 contractions in 10 minutes).
According to NCC and AWHONN standards, when tachysystole is present with minimal variability, oxytocin must be reduced or discontinued even in the absence of late decelerations.
Clinical decision-making (per NCC principles):
NCC emphasizes that management of Category II patterns during induction starts with intrauterine resuscitative measures, including decreasing or stopping oxytocin when uterine activity is excessive or fetal response is suboptimal. Minimal variability with tachysystole requires correction of uterine stimulation before escalating to invasive monitoring or considering operative birth.
Option B (place a spiral electrode) is not indicated because the pattern is clearly visible and the priority is correcting uterine overstimulation, not refining the tracing.
Option C (operative birth) is not indicated; there is no Category III pattern or recurrent decelerations.
Option A (discontinue oxytocin) is the correct first-line action according to NCC-aligned guidelines when tachysystole and minimal variability occur.
References:
NCC C-EFM Candidate Guide (2025); NCC Content Outline; NICHD Three-Tier FHR Interpretation System; AWHONN Fetal Heart Monitoring Principles & Practices; Miller's Fetal Monitoring Pocket Guide; Menihan Electronic Fetal Monitoring; Simpson & Creehan Perinatal Nursing; Creasy & Resnik Maternal-Fetal Medicine.

NEW QUESTION # 105
Interventions undertaken to address fetal tachycardia are targeted at maximizing
  • A. maternal circulation
  • B. uteroplacental perfusion
  • C. sympathetic autonomic tone
Answer: B
Explanation:
Comprehensive and Detailed Explanation From Exact Extract NCC-Recommended Sources Fetal tachycardia is typically caused by maternal fever, dehydration, hypoxia, medications, infection, or fetal stress. AWHONN and Simpson & Creehan emphasize that management focuses on improving oxygen delivery across the placenta, which is governed by uteroplacental perfusion.
Menihan's EFM text states that "interventions for fetal tachycardia must address oxygen transfer by optimizing uteroplacental blood flow," including hydration, reducing uterine activity, maternal repositioning, and treating maternal fever.
Increasing maternal circulation alone is insufficient unless it improves placental blood flow. Enhancing fetal sympathetic tone is not a clinical goal and would worsen tachycardia.
Creasy & Resnik highlight that fetal heart rate abnormalities resolve when uteroplacental perfusion is restored, confirming this as the primary target of intervention.
References:
AWHONN - Fetal Heart Monitoring Principles & PracticesSimpson & Creehan - Perinatal NursingMenihan
- Electronic Fetal MonitoringCreasy & Resnik - Maternal-Fetal MedicineMiller's Pocket Guide

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