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[Hardware] Test EFM Engine Version - EFM Exam Learning

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【Hardware】 Test EFM Engine Version - EFM Exam Learning

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The competition in the NCC field is rising day by day and candidates around the globe are striving to validate their capabilities. Because of the rising competition, candidates lack opportunities to pursue their goals. That is why has launched the NCC EFM Exam to assess your capabilities and give you golden career opportunities. Getting a Certified - Electronic Fetal Monitoring (EFM) certification after passing the NCC EFM exam is proof of the capabilities of a candidate.
ExamsLabs is the door to success. ExamsLabs NCC EFM Test Questions are very similar to the actual test. At the same time, our ExamsLabs NCC EFM test questions and test answers are studied, based on the same syllabus. And we are also constantly upgrading our training materials. So, pass rate is 100% and high quality guarantee!
EFM Exam Learning, Valid Dumps EFM SheetThe NCC EFM practice test software also keeps a record of attempts, keeping users informed about their progress and allowing them to improve themselves. This feature makes it easy for EFM desktop-based practice exam software users to focus on their mistakes and overcome them before the original attempt. Overall, the Windows-based Certified - Electronic Fetal Monitoring (EFM) practice test software has a user-friendly interface that facilitates candidates to prepare for the NCC EFM exam without facing technical issues.
NCC Certified - Electronic Fetal Monitoring Sample Questions (Q58-Q63):NEW QUESTION # 58
A woman at 41-weeks gestation is being induced. She is 2 cm dilated and is on oxytocin at 8 milliunits
/minute. Based on the fetal heart rate tracing shown, the best initial response is to:

  • A. Decrease the oxytocin
  • B. Place a fetal spiral electrode
  • C. Continue to observe
Answer: A
Explanation:
Comprehensive and Detailed Explanation From Exact Extract-Based NCC C-EFM References:
The tracing shows tachysystole with emerging late decelerations and minimal variability:
* 5 contractions in 10 minutes
* Deceleration nadirs occur after the peak of the contraction (late pattern)
* Variability begins to trend toward minimal
* The tracing has deteriorated while on oxytocin 8 mU/min, a common threshold for overstimulation NCC and AWHONN emphasize that when tachysystole occurs with any fetal intolerance, the first action is to reduce or stop oxytocin.
Key NCC principles:
* Late decelerations + tachysystole = uteroplacental insufficiency caused by excessive uterine activity
* Interventions:
* Stop or reduce oxytocin
* Maternal repositioning
* IV fluid bolus
* Possible oxygen if other measures fail
Why the other options are incorrect:
* A. Continue to observe - not acceptable with late decels + tachysystole.
* C. Place a spiral electrode - this corrects signal quality, not uterine overstimulation or fetal oxygenation.
Thus, the best initial response is B. Decrease the oxytocin.
References:NCC C-EFM Candidate Guide; AWHONN Fetal Heart Monitoring Principles & Practices; NICHD Definitions; Miller & Menihan EFM texts; Simpson & Creehan; Creasy & Resnik.

NEW QUESTION # 59
Interventions undertaken to address fetal tachycardia are targeted at maximizing
  • A. maternal circulation
  • B. uteroplacental perfusion
  • C. sympathetic autonomic tone
Answer: B
Explanation:
Comprehensive and Detailed Explanation From Exact Extract NCC-Recommended Sources Fetal tachycardia is typically caused by maternal fever, dehydration, hypoxia, medications, infection, or fetal stress. AWHONN and Simpson & Creehan emphasize that management focuses on improving oxygen delivery across the placenta, which is governed by uteroplacental perfusion.
Menihan's EFM text states that "interventions for fetal tachycardia must address oxygen transfer by optimizing uteroplacental blood flow," including hydration, reducing uterine activity, maternal repositioning, and treating maternal fever.
Increasing maternal circulation alone is insufficient unless it improves placental blood flow. Enhancing fetal sympathetic tone is not a clinical goal and would worsen tachycardia.
Creasy & Resnik highlight that fetal heart rate abnormalities resolve when uteroplacental perfusion is restored, confirming this as the primary target of intervention.
References:
AWHONN - Fetal Heart Monitoring Principles & PracticesSimpson & Creehan - Perinatal NursingMenihan
- Electronic Fetal MonitoringCreasy & Resnik - Maternal-Fetal MedicineMiller's Pocket Guide

NEW QUESTION # 60
A characteristic of early decelerations is that they
  • A. are thought to be caused by a vagal reflex
  • B. commonly fall below 100 beats per minute
  • C. are episodic
Answer: A
Explanation:
Comprehensive and Detailed Explanation From Exact Extract (No URLs or Links):
Early decelerations are defined in NCC and AWHONN resources as gradual, uniform decelerations that mirror uterine contractions and are associated with fetal head compression. AWHONN's Fetal Heart Monitoring Principles states: "Early decelerations are a benign pattern caused by vagal stimulation secondary to fetal head compression." Menihan similarly notes: "The mechanism of early decelerations is a vagal reflex response; they do not reflect hypoxia." They are periodic, not episodic, because they occur with contractions-which rules out option A.
They typically remain within a normal heart rate range and do not usually fall below 100 bpm; this eliminates option C. NCC Candidate Guide emphasizes that early decelerations are considered a normal physiologic response, not a pathologic pattern, and are categorized as "Category I" when variability is present.
Thus, the correct characteristic is that they are caused by a vagal reflex, making B the correct answer.
References:AWHONN Fetal Heart Monitoring ProgramMenihan: Electronic Fetal MonitoringSimpson & Creasy: Fetal PhysiologyNCC C-EFM Content Domains - Physiology

NEW QUESTION # 61
A woman is admitted at 41-weeks gestation for fetal evaluation following a motor vehicle accident. She reports that she hit her abdomen on the steering wheel. The underlying physiology of the tracing is most likely:

  • A. Placental abruption
  • B. Fetal trauma
  • C. Cord accident
Answer: A
Explanation:
Comprehensive and Detailed Explanation From Exact Extract-Based NCC C-EFM References:
This tracing shows recurrent late decelerations, decreased variability, and subtle baseline shifts-findings that strongly correspond to uteroplacental insufficiency. In trauma cases, NCC emphasizes that placental abruption is the most common fetal complication, caused by shearing forces separating the placenta from the uterine wall.
Key physiologic points per NCC/AWHONN/Menihan:
* Maternal blunt abdominal trauma frequently leads to partial or concealed abruption.
* Abruption produces decreased uteroplacental blood flow, resulting in:
* Late decelerations
* Minimal/absent variability
* Baseline shifts or instability
Cord accident (option A) typically produces variable decelerations, not late-pattern decelerations.
Fetal trauma (option B) is extremely rare and does not produce a consistent deceleration pattern.
Thus, the physiology most consistent with this tracing and mechanism of injury is placental abruption.
References:NCC C-EFM Candidate Guide (2025); NCC Physiology Domain; AWHONN Fetal Heart Monitoring Principles & Practices; Menihan Electronic Fetal Monitoring; Simpson & Creehan Perinatal Nursing; Creasy & Resnik Maternal-Fetal Medicine.

NEW QUESTION # 62
A fetal heart rate tracing is abnormal. A change in maternal position and oxygen administration do not correct the pattern. Following birth, a fetal cord blood sample is taken:
pH = 7.25
PaCO# = 46 mm Hg
PaO# = 20 mm Hg
HCO# = 22 mEq/L
Base deficit = -4 mEq/L
These results are best interpreted as:
  • A. Acidosis
  • B. Hypoxia
  • C. Normal
Answer: C
Explanation:
Comprehensive and Detailed Explanation From NCC-Aligned Sources:
Normal umbilical arterial values per NCC/AWHONN/Menihan:
* pH: 7.20-7.30
* PaCO#: 45-55 mmHg
* HCO#: 20-24 mEq/L
* Base deficit: 0 to -5 (normal to mild respiratory changes)
This sample shows:
* pH 7.25 # normal
* Base deficit -4 # no metabolic acidosis
* HCO# normal
* Slightly elevated PaCO#, consistent with mild respiratory influence but still normal
* PaO# 20 mmHg is normal for cord arterial blood
This profile is not acidotic (acidosis requires pH <7.10 and base deficit #12).
It also does not indicate hypoxia, which would present with metabolic acidosis.
Therefore: Normal.
References:NCC C-EFM Candidate Guide; AWHONN FHMPP; Menihan; Simpson & Creehan; Creasy & Resnik.

NEW QUESTION # 63
......
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